Max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. Alternatively, our capability to rescue SERT functionality in KI midbrain synaptoneurosomes from the inhibition of FAK indicates elevated FAK signaling downstream with the Pro32Pro33 mutant, as verified by elevated pFAK localization in 5-HT synapses. Our data https://martinn642pbl3.plpwiki.com/user