Max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. On the other hand, our ability to rescue SERT functionality in KI midbrain synaptoneurosomes via the inhibition of FAK implies elevated FAK signaling downstream with the Pro32Pro33 mutant, as verified by improved pFAK localization in 5-HT https://kameronpdpyj.buyoutblog.com/32307533/the-definitive-guide-to-pro33