Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. Then again, our capacity to rescue SERT purpose in KI midbrain synaptoneurosomes because of the inhibition of FAK suggests elevated FAK signaling downstream with the Pro32Pro33 mutant, as verified by elevated pFAK localization in 5-HT synapses. https://posecio642rcn3.idblogz.com/profile